- Information
- Symbol: CycB1;1,OsCycB1;1
- MSU: LOC_Os01g59120
- RAPdb: Os01g0805600
- PSP score
- LOC_Os01g59120.1: 0.0476
- PLAAC score
- LOC_Os01g59120.1: 0
- pLDDT score
- 74.53
- Protein Structure from AlphaFold and UniProt
- Publication
- Genbank accession number
- Key message
- Furthermore, resistance to cold stress in the transgenic plants overexpressing OsCycB1;1 was also enhanced
- We show that OsMYB3R-2 specifically bound to a mitosis-specific activator cis-element, (T/C)C(T/C)AACGG(T/C)(T/C)A, a conserved sequence that was found in promoters of cyclin genes such as OsCycB1;1 and OsKNOLLE2
- These results suggest that OsMYB3R-2 targets OsCycB1;1 and regulates the progress of the cell cycle during chilling stress
- Expression analysis showed that Orysa;CycB1;1 transcripts were localized in the endosperm and embryo
- The results of the knockdown of Orysa;CycB1;1 via an endosperm or an embryo-specific promoter also suggest that the enlarged embryo may be correlated to the abortive endosperm
- Our results suggest that Orysa;CycB1;1 expression is critical for endosperm formation via the regulation of mitotic division, and that the endosperm plays an important role in maintenance of embryo development in rice
- The expression of Orysa;CycB1;1 is essential for endosperm formation and causes embryo enlargement in rice
- In our current study, we show that the knockdown of Orysa;CycB1;1 in rice results in the production of abnormal seeds, which at maturity contain only an enlarged embryo
- Connection
- CPT1, CycB1;1~OsCycB1;1, Enhanced tolerance to chilling stress in OsMYB3R-2 transgenic rice is mediated by alteration in cell cycle and ectopic expression of stress genes, OsCPT1, which may be involved in the dehydration-responsive element-binding factor 1A pathway, showed the same transcription pattern in response to cold as did OsCycB1;1 in transgenic rice
- CycB1;1~OsCycB1;1, OsMYB3R-2, Enhanced tolerance to chilling stress in OsMYB3R-2 transgenic rice is mediated by alteration in cell cycle and ectopic expression of stress genes, We show that OsMYB3R-2 specifically bound to a mitosis-specific activator cis-element, (T/C)C(T/C)AACGG(T/C)(T/C)A, a conserved sequence that was found in promoters of cyclin genes such as OsCycB1;1 and OsKNOLLE2
- CycB1;1~OsCycB1;1, OsMYB3R-2, Enhanced tolerance to chilling stress in OsMYB3R-2 transgenic rice is mediated by alteration in cell cycle and ectopic expression of stress genes, In addition, overexpression of OsMYB3R-2 in rice led to higher transcript levels of several G2/M phase-specific genes, including OsCycB1;1, OsCycB2;1, OsCycB2;2, and OsCDC20
- CycB1;1~OsCycB1;1, OsMYB3R-2, Enhanced tolerance to chilling stress in OsMYB3R-2 transgenic rice is mediated by alteration in cell cycle and ectopic expression of stress genes, The level of cellular free proline was increased in the overexpressed rice lines of OsMYB3R-2 and OsCycB1;1 transgenic plants compared with wild-type plants under the cold treatment
- CycB1;1~OsCycB1;1, OsMYB3R-2, Enhanced tolerance to chilling stress in OsMYB3R-2 transgenic rice is mediated by alteration in cell cycle and ectopic expression of stress genes, These results suggest that OsMYB3R-2 targets OsCycB1;1 and regulates the progress of the cell cycle during chilling stress
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