- Information
- Symbol: OsIBH1,IBH1
- MSU: LOC_Os04g56500
- RAPdb: Os04g0660100
- PSP score
- LOC_Os04g56500.1: 0.1584
- PLAAC score
- LOC_Os04g56500.1: 0
- pLDDT score
- 76.61
- Protein Structure from AlphaFold and UniProt
- Publication
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Genbank accession number
- Key message
- Overexpression of ILI1 or PRE1 increases cell elongation and suppresses dwarf phenotypes caused by overexpression of IBH1 in Arabidopsis
- ILI1 and PRE1 interact with basic helix-loop-helix (bHLH) protein IBH1 (ILI1 binding bHLH), whose overexpression causes erect leaf in rice and dwarfism in Arabidopsis
- BR increases the RNA levels of ILI1 and PRE1 but represses IBH1 through the transcription factor BZR1
- The spatial and temporal expression patterns support roles of ILI1 in laminar joint bending and PRE1/At IBH1 in the transition from growth of young organs to growth arrest
- Connection
- ILI1, OsIBH1~IBH1, Antagonistic HLH/bHLH transcription factors mediate brassinosteroid regulation of cell elongation and plant development in rice and Arabidopsis, ILI1 and PRE1 interact with basic helix-loop-helix (bHLH) protein IBH1 (ILI1 binding bHLH), whose overexpression causes erect leaf in rice and dwarfism in Arabidopsis
- ILI1, OsIBH1~IBH1, Antagonistic HLH/bHLH transcription factors mediate brassinosteroid regulation of cell elongation and plant development in rice and Arabidopsis, Overexpression of ILI1 or PRE1 increases cell elongation and suppresses dwarf phenotypes caused by overexpression of IBH1 in Arabidopsis
- ILI1, OsIBH1~IBH1, Antagonistic HLH/bHLH transcription factors mediate brassinosteroid regulation of cell elongation and plant development in rice and Arabidopsis, BR increases the RNA levels of ILI1 and PRE1 but represses IBH1 through the transcription factor BZR1
- ILI1, OsIBH1~IBH1, Antagonistic HLH/bHLH transcription factors mediate brassinosteroid regulation of cell elongation and plant development in rice and Arabidopsis, The spatial and temporal expression patterns support roles of ILI1 in laminar joint bending and PRE1/At IBH1 in the transition from growth of young organs to growth arrest
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